Portrait Artist and Silk Painter

My Diet IS STRICT: No sugar (I use Stevia as sweetener (trade name Truvia). No carbohydrates, No bread, No fruit except blueberries & homegrown black currents. No tomatoes. No beetroot. No sweet potatoes etc. If you are 'addicted' to sugar and carbs, this diet is not for you! Side effect: I eat lots - but lose weight.  

Below: Typical evening meal with organic broccoli, cauliflower, mushrooms, green beans etc., a little chicken, or fish. Sometimes a little liver or red meat (organic when I can get it). 

I turned to Facebook Groups for help.

There are many specific cancer groups. In December 2024 I explained my predicament to members of the ‘Healing Cancer Study Support Group’. 100+ members replied. I knew nothing about cancer – so carefully noted everything and then looked up/researched their suggestions.

One of the most important suggestions (made several times) was to read 

Jane McClelland’s Book, How to Starve Cancer. 

I am a mother, grandmother, artist and author. I follow my own gut instincts, and chemotherapy, especially very high platinum doses, seemed completely against my instinct. 

Why destroy your health to destroy the cancer cells? Is there a better way? 

Can I destroy cancer cells and remain healthy? 

We are fortunate today to have accessible online libraries where worldwide research papers are filed. If you ask Professor Google a question, or ask ChatGPT, they are brilliant at hunting out research and trials. 

Facebook can be brilliant especially if you find curated groups with no ‘quack’ doctor rubbish. So many organisations, pharma companies, quack doctors etc., try to make money out of illness and cancer, telling you of  miracle cures etc.  Personally, I don’t think there is a ‘miracle’ cure for my disease. I have to keep working at it with a strict diet and variety of medications/herbs etc., to try and slow progression and maybe improve. 

I am monitoring what I am doing and publishing the results. 

Most blood tests and scans, I am now self-funding. UK doctors & oncologists have strict 'rules' and cannot step outside of these and most seem quite uninterested in any other type of possible therapies. I pay for most of my own tests so I can monitor my progression. 

I ask for no contribution or donation for sharing my research.

If you would like to show your appreciation, please order a copy of the ENCHANTED CASTLE by S.K. Moore. (from Amazon Books). Details on the home page. I wrote and illustrated it with my son to help him to read. It won a Best Children’s Book of the Year award. Now republished, with drawings that can be coloured in, and page layouts designed with dyslexia in mind. 

Proceeds from the sale of this book will be donated to children’s charities. 

CA125 BLOOD TEST RESULTS

 November 2024 diagnosed with stage three high grade serous ovarian cancer. HGSOC

 19th Nov. 2024 CA125 blood test = 345 – started vegan diet and began research.

25^th Feb. 2025 CA125 =   698 (after 1 cycle of carboplatin chemo 5^th Feb.2025)

 Mid Mar. 2025 CA125 =    645 vegan diet, plus anti-cancer supplements – details below

7^th April 2025   CA125 =   230.1 (taking RSO THC cannabis for 3-month trial)

24^th April 2025 CA125 =    97.2 (taking RSO THC cannabis vaginal suppositories 1 month)

8^th May 2025    CA 125 =   115.1 (taking RSO THC cannabis)

27^th June 2025 CA125 =   155.3 (taking RSO THC cannabis – end of three-month trial)

17^th July 2025 CA 125 = 166 (taking herbs, aspirin, Fenbendazole, Ivermectin etc). (NHS)

17^th July 2025 – NHS blood test to check potassium, kidneys etc. All okay but slightly anemic. 

SCANS

21^st Nov. 2024 ULTRASOUND

Within the midline of the pelvis and extending into both adnexa there was a large irregular multilocular lesion 15.5 x 11 x 7.5 cm. Contained both solid and cystic components. Likely to be malignant. 

6^th June 2025. ULTRASOUND 

(paid for privately as not being supported by my GP) Large irregular partly solid partly cystic tumour sited in the pelvis crossing the midline. 10.2 x 8.1 x 5.8 cm. The mass displaces the uterus anteriorly which is rotated to the right. The endometrium is thin and linear. Mild volume of anechoic fluid in the POD, 16 x 15 x 17 mm. The right mass is 4.9 x 4.8 x 4.8xm. Left 8.3 x 5.8 x 4.7 cm. There is papillary growth inside and outside the cystic cavity. There is colour flow within the mass.

CT SCAN 21^st Nov. 2024

The urinary bladder appeared ultrasonically normal. Uterus was retroverted and normal in size and outline. The myometrium appeared homogenous in echotexture. The endometrial midline appeared uniform with a thickness of 1.5 mm at the fundus.

Within the midline of the pelvis and extending into both adnexa there was large irregular multilocular lesion 15.5 x 11 x 7.5 cm. Both solid and cystic components and a strong increase in vascularity (IOTA – M4 and M5). No free fluid evident.

(Report edited):

Bilateral adnexal mass, solid cystic with peripheral nodules and internal septation. Both lesions are extending to pelvis side wall. Right measures 6.4 x 6 x 5.4 cm. Left measures 16 x 11 x 10 cm 

One cycle of Carboplatin 5^th February 2025. (I refused platinum chemo with 2 more chemo’s, so I kept my hair!). Two weeks of feeling very unwell, affected my right kidney and I became anemic. I cancelled further chemo as too toxic. 

My GP and Oncologist have refused to support me as I would not accept further chemotherapy. I mostly pay privately for CA 125 blood tests and ultrasound scan.

Cancer Stem Cells are not usually killed by chemotherapy. Biopsies may further ‘spread’ the cancer. When tumours are burst by a needle being poked into them, the cells can escape.

 My ambition is to learn to live with my cancer.

I take various things – listed below, which have proven in the petri dish, and in mice, they can ‘kill’ ovarian cancer cells. Sadly, there are no human trials. So, I am a human guinea pig  trying to slow down the progression. (Many of these things also kill other types of cancer but as each cancer is different, please do your own research.)

RESULTS OF MY GUINEA PIG ‘TRIALS’ 

1)    November 2024 to February 2025. Tried to follow Gerson Therapy: Diet, Food, enemas, juicing vegetables etc. No sugars, low carbs etc.

Result: CA125 blood test continued to increase from 345 to 545 pre chemo, so I don’t think this helped, although it may have slowed progression. 

2)    One cycle of Carboplatin chemotherapy 5^th February 2025.

Result: Made me ill. Kidney pain and became anemic. CA125 went up to 698.

 

3)    March 2025 – June 2025. Vegan diet and started to take RSO THC plus supplements listed below.

Result:  Lowered my CA125 blood results and tumours shrunk by approx. one third. // RICK SIMPSON OIL is strong THC cannabis. Taken both orally, and one month of vaginal suppositories. Yes, I think this had a big effect on reducing my tumour sizes. I followed this protocol (plus the herbals, repurposed drugs etc, listed below), for three months from mid-March to mid-June. Hated it as it numbed my brain and made me very tired.

 4)    JUNE 2025 – changed to KETO diet. No carbs, no sugars, etc. Continuing with complimentary herbals, Increased Ivermectin twice a day, 4 days on, 2 days off.  Fenbendazole 4 days on 3 days off. Aspirin. Loratadine etc.

Result: 17^th July 2025 CA125 = 166. Slight increase, so this may be slowing progression, but probably not reducing the size of my tumours. 

Example of my diet (1) 

November to June 2025

Platefuls of vegetables, broccoli, cauliflower, mushrooms, celery, courgettes, green beans, avocados, olive oil, walnuts, almonds, cashews, organic peanut butter with Fenbendazole, garlic, black garlic etc. One slice of gluten free home-made seeded bread with olive oil. Occasionally - one piece of dark 85% chocolate after my meal. Occasionally a small piece of meat or fish and one egg per week. 

(2) 5. JULY 19^th onwards. Continuing with Keto diet and herbals etc listed below. Will reduce Fenbendazole. Starting to take a statin, Atorvastatin. Has shown to have potential anti-cancer effects, including against ovarian cancer, in both in vitro and in vivo studies. My GP, quite rightly, would not prescribe this as my cholesterol levels are low: 2.8. (Dec 2024). However, a friend has provided them. Having very, very, low cholesterol levels is not good – but I am cautiously taking a risk for three weeks to see if I can lower my CA125.  

Absolutely no sugars, no carbs i.e. no wheat bread, no dairy except sometimes a little double cream as a sauce with my dinner, and sometimes a little goat’s yoghurt. No cakes, no biscuits, no fruits (except blueberries and homegrown blackcurrants). Fruits are high sugar and convert into blood sugars which the cancer cells like to live on. Drinks: filtered water, ginger & lemon tea, fruit tea with Hawthorne Berry powder, one cup of organic coffee with a little soya milk. 

(2) FROM JUNE 2025 – changing from Vegan to KETO DIET

(This is easier to manage if you are on holiday or eating in a restaurant.) 

70% vegetable as above, 30% meat or fish. (Chips and peas do not count as ‘vegetables). My body is now in Ketosis – whereby I am hoping to starve my cancer cells but not my normal cells! Please do your own research for more information on what you can and cannot eat. I probably eat less than 30% meat/fish. Still mainly vegetarian.

SIDE EFFECT: Night time leg/foot cramps. Waking me up every hour through the night, probably due to low blood sugar levels. (Blood tests 17^th July – checked potassium levels, magnesium etc. All okay). As I have lost weight since being diagnosed my muscle strength is not so good, and I do get tired. Otherwise, I feel healthy and well. 

Conflicting Information about Keto diet edited from FACEBOOK by LAURA KRZEWINSKI LANG 

All cells use glucose for energy. Many cancers ae driven by fat. See Triple negative breast cancer. It is fat that drives it. 

KETOSIS – Keto diet. The problem with keto is you are not getting your polyphenols and other antioxidants that help to heal. 

You have to make your body think it is starving before you can get into ketosis.  If you are low carb your liver will be signalled to steal fat from your muscles, go into gluconeogenesis. 

Another big driver of cancer is IGF-1 (growth) hormones. They contribute to cancer growth. People who eat animal foods, including fish, will have a much high IGF-1 hormone level. 

It only takes about 8 weeks on a plant-based diet to dramatically reduce IGF-1 hormones which do drive cancer growth. Please do NOT attempt ketosis. It is unnatural and only used by the body to prevent cell death leading to body death. 

Also eat to keep your body from metabolizing TMAQs to avoid endothelial damage.

The gut microbiome is 70% of your immune system. 

If you don’t eat 30-40- grams of fibre a day, your gut microbiome has no food to eat. Instead, it will eat the protective mucosal lining of your blood vessels.

 *My own note about being vegan / vegetarian. After six months It did not reduce my cancer CA125 results. I am now trying KETO and will see if this has a positive or negative result.

 LIST OF SUPPLEMENTS BELOW  

These are not a recommendation but a note of things I take. I have given my list of ‘complimentary supplements’ to two different oncologists, who both said that what I was taking shouldn’t be harmful. 

As a ‘human’ guinea pig, I try to work out (guess) what dose to take. From the quantities they give mice, to the quantity a human should take, is a guess. I am naturally cautious not to take too much of anything that may be harmful to me.

All cancers are different. What works on mice may not work on humans. Frustratingly, NO human trials are currently being done by either Cancer Charities, or big pharma companies into alternative therapies.  Maybe no one is interested in a ‘cure’ unless they can make money?

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ANGELICA SINENSIS Dong Quai (organic powder- supplier Indigo Herbs). N-butylidene phthalide kills high grade serous ovarian stem cells by activating intrinsic apoptosis signalling pathways. //pmc.ncb.nim.nih.gov

Taking heaped teaspoon in hot water once or twice a day. Tastes ‘horrid’. Conflicting research on this. Some research says do not take for hormone driven cancers (i.e. breast & ovarian cancer.) My cancer is not hormone driven. Note: stopped taking this in May (diluted in liquid/water) as tastes horrid. //

July 19^th 2025, resumed taking – but disguising taste in a ‘pancake’.  

Recipe: 2 or 3 tablespoons of Ground Almonds/flour (from Sainsbury). 1 teaspoon Dong Quai. Half teaspoon of dried ginger and Stevia (Truvia), Quarter teaspoon of bicarbonate of soda. Mix together with soya milk to paste. Spoon out and fry gently in small pancake sizes in olive oil for about 5 minutes.    

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Aspirin 75 mg coated. Inhibits cell proliferation and induce apoptosis in various cancer cell lines. Helps prevent metastasis. I take one tablet daily with food/evening meal. Most cancer patients do not die from initial tumour, but from metastasis i.e. the spread of cancer to other organs. Aspirin helps to prevent this. I take one each evening with my meal.

 Blueberries – polyphenolic acids, and soy isoflavone genistein derivative, specific inhibition of ovarian Cancer Stem Cells. (pmc.ncb.nim.nih.gov National Institute of Health). (Best to eat organic and wild blueberries)

 Broccoli eaten raw, cooked, and sprouted seeds. (Sulforaphane, cancer-fighting plant compound). Most days I eat half to three quarters of a broccoli. Also, half a cauliflower, raw or cooked, or roasted with sesame seeds. (I enjoy raw cauliflower with my breakfast.)

 Black Garlic – fermented form of garlic – has many bioactive compounds that give it medicinal properties, including ant- inflammatory and anti-cancer properties.  Reduction of pro-inflammatory cytokines, ability to scavenge free oxygen radicals and induce apoptosis. Antiproliferative and antiangiogenic properties and inhibit the growth of cancer cells. (Not effective on some breast and lung cancer). (I buy it from Sainsburys and Ocado). It tastes quite sweet, and does not make your breath smell quite so badly as fresh garlic.

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Berberine (METFORMIN alternative) – 1 capsule before each meal. To lower blood glucose and increase insulin sensitivity. (From British Supplements 422 mg Amur Cork tree Extract.) (In vitro experiments, berberine reduces glycolysis and autophagy levels, leading to the inhibition of ovarian cancer cell proliferation and metastasis.) www.sciencedirect.com  Berberine can take about 3 months to lower blood sugar. Metformin works much faster in 4-5 days.  Research shows combination of berberine, and metformin can enhance the effects on blood sugar. Berberine can make it easier to tolerate metformin with fewer metformin side effects. Berberine shown to regulate both glucose metabolism and lipid metabolism. Berberine did not cause changes in kidney or liver function tests. Powerful oral blood sugar-lower with beneficial effects on fasting insulin and lipid metabolism. Berberine starting dose (for diabetes)300 - 500 mg per day, increasing to 3 times a day. Absorption in the gastrointestinal tract can be enhanced by taking milk thistle. Metformin and berberine can cause flatulence, diarrhea, constipation and abdominal pain. 

 Bicarbonate of Soda (Baking soda). Cancer tumours are acidic. Bicarb is alkaline. An alkaline environment might be anti-cancer – but not enough ‘human’ research done. I take a small amount in my morning coffee and take a bicarb capsules when I remember with my drinks. Research exploring anti-cancer effects began in 1990s. Several in vivo experiments revealed potential anticancer effects. Sodium bicarbonate neutralises the acidity and increases the tumour PHe and might control tumour progression. If injected directly into the tumour, may kill the cancer cells without chemo, but human trials have not been done. 

Cordycepin (fungus) Induces apoptosis of human ovarian cancer cells.  Taking 1 or 2 capsules per meal. (Supplier: British Supplements. 488 mg Cordyceps extract, 195 mg Polysaccharides (can lower cholesterol). Adenosine 4.8 mg

Cordycepin (fungus) also known as 3-deoxyadenosine. Contains various bioactive ingredients, suggested to have anti-cancer potential. Adenosine derivatives from Cordyceps exert antitumour effects against ovarian cancer cells through ENT1-mediated transport, induction of AMPX signalling and consequent autophagic cell death. https://www.sciencedirect.com

CORDYCEPS MILITARIS Results suggest that Cordyceps militaris induces apoptosis in ovarian cancer cells through TNF-a/TNFR1-mediated inhibition of NF-kB phosphorylation. I

Exhibits pharmacological activities, including antitumour properties through the regulation of the nuclear factor kappa B (NF-mB) signalling. Tumour Necrosis Factor (TNF) and TNF0a modulates cell survival and apoptosis through NF-kB signalling. However, the mechanism underlying its mode of action of the NF-kB pathway is unclear.

Results suggest that Cordycepin Militaris inhibited ovarian cancer cell proliferation, survival, and migration, possibly through coordination between TNF-a/TNFR1 signalling and NF-kB activation. Taken together, our findings provide a new insight into a novel treatment strategy for ovarian cancer using Cordycepin Militaris.  National Library of Medicinemed.ncbi.nim.nih.gov

*Could interact with blood thinners and drugs that suppress the immune system. Also cause problems if you take medications to lower your blood sugar levels.

 DOXYCYCLINE - Tetracycline Antibiotic. (Anti malaria treatment).  Inhibits proliferation of epithelial ovarian cancer cells. Taking 1 capsule 100 mg mid-morning with lots of water, 3 days on 1 day off. Can make me sick! (Stopped taking this in June 2025 as experimenting with increasing my dose of Ivermectin)

DOXYCYCLINE  - (From Online Pharmacy). (to prevent malaria and treat infections caused by mites, ticks or lice. etc). Exhibits anti-inflammatory anti-tumour, and pro-apoptotic activity. Is being tested as chemotherapeutic agent for several cancers. Inhibits proliferation of epithelial ovarian cancer cells. (Used with cisplatin and doxycycline in different concentrations, cell viability was decreased in a dose dependent manner). https//pmc.ncbi.nim.gov. Avoid eating dairy, eggs, kale and spinach. It aims to slow down or kill bacteria by inhibiting its protein production and can induce a number of side effects such as diarrhoea, nausea, vomiting.

Doxycycline, Azithromycin and Vit C (DAV) (National Library of Medicine pmc.ncbi.nim.nih.gov ).

A potent combination therapy for targeting mitochondria and eradicating cancer stem cells.  Trial conducted University of Pisa Hospital, aimed to assess the anti-proliferative and anti-CSC mechanistic actions of Doxycycline in early breast cancer patients. Promising results. Preliminary effects of Doxycycline on CSC propagation and employing a combination therapy with Azithromycin and Vit C.  Azithromycin inhibits the large mitochondrial ribosome. Vit C acts as a mild pro-oxidant, which can produce free radicals, and as a consequence, induces mitochondrial biogenesis.

Results indicate that a combination is more efficacious than the individual antibiotic in inhibiting CSCs propagation.

Doxycyline inhibits the small mito-ribosome .

Azithromycin blocks function of the large mito-ribosome as an off-target effect.

(Note: combination may affect your gut biome.) 

 Fenbendazole I started taking this February 2025. 4 days on, 3 days off (to protect my liver). Animal worming and parasite medication for animals. (Kills worms by reducing their sugar/glucose uptake.) Mebendazole is the ‘human’ equivalent with a prescription. Taking 1 capsule per 10 kilos in weight. e.g. 5 x 50 mg capsules mixed with peanut butter and olive oil (No drinks with it as it is not water soluble). Available to order from Etsy.

FENBENDAZOLE – Joe Tippens in 2016 was diagnosed with small cell lung cancer, one of the most aggressive types of cancer. It had spread throughout his body. He had gone through conventional treatment and was preparing for the end. However, someone said to try Fenbendazole. He was ‘cured’ and his story spread across the world to other cancer patients. 

Mixed reviews on anti-cancer treatment for humans. (Many recommendations from people living with cancer). Mebendazole is the similar ‘human’ medication, but not available without a prescription. 

Research: Oral admin of FBZ inhibited tumour growth in a mouse model of xenograft ovarian cancer. FBZ has therapeutic potential for the treatment of ovarian cancer. bmccancer.biomedcentral.com 

Exhibits several other mechanisms contributing to its anti-cancer effects, primarily by disrupting energy metabolism. https://ar.liarjournals.org Anticancer Research

  IVERMECTIN (ANIMAL AND HORSE anti-parasite drug)

 can suppress almost completely the growth of various human cancers, including ovarian cancer.  I took a small amount most days from March onwards. Personally, I don't think it reduced my cancer, but it may have helped maintain it at a lower level. Working out the dose, between an animal and human is difficult, and we cannot be sure how much 'mice' were given to reduce their cancer.

July 2025 3-week trial, I increased dose to twice, sometimes three times per day. Either half an hour before eating and 2 hours after eating.

Side effect – saw turquoise polka dots, and swirls when I closed my eyes. Gave myself a 2-day break before resuming experiment and was okay again.  My CA125 result much the same as the previous month at 166, so did not reduce my cancer. (From 20^th July, also taking a statin and the combination may be more effective.) 

IVERMECTIN  - Anti-parasite drug can suppress ovarian cancer by regulating IncRNA-EIF5A3-mRNA axes. Its anticancer mechanism remains to be further studied at molecular levels. National library of Medicine. pmc.ncbi.nim.nih.gov 

Used to treat River Blindness and other parasitic diseases. Has saved millions of lives especially in the developing world.

IVERMECTIN is believed to be safe and in much of the civilized world it is available over the counter, no prescription needed. However, in the UK it is not available and may be obtained from some (horse) suppliers if you have a horse or two! e.g. Hyperdrug.co.uk

Used as pesticides, insecticides and anti-parasitic. Inhibits glutamate-activate chloride channels. Inhibits pathways etc. 

Recommended Doses:

·         Low Dose 0.5 mg per kilo of body weight. Dr Tess Lawrie reported case of Stage 3 Ovarian Cancer, treated with chemo and 12 mg of IVM date. CA125 dropped 288 to 22 after 2 months. Tumour vanished.

·         Medium Dose: 1.0 mg per kilo body weight.  Starting dose for most cancers. No long-term side effects.

·         High Dose: 2.0 mg per kilo body weight. For very aggressive cancers. No long-term side effects.

·         Very High Dose: 2.5 mg per kilo body weight. For Extensive metastatic disease with poor prognosis. Possible short term and transient visual effects. Dr Shankara Chetty had a patient on this with no side effects.

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 LORATADINE antihistamine - cationic amphiphilic (CAD) Induces cancer-specific cell death in experimental studies. (Good for lung cancer). I take 1 capsule with evening meal most days. It is easily available from UK chemists to treat hayfever etc. 

LORATADINE Reduction of around 20%-35% in ovarian cancer mortality. CADs a diverse group of compounds, accumulate in acidic lysosomes where they can induce permeabilization of the lysosomal membrane leading to cell death. The cancer cells, as opposed to normal cells, lysosomes are more abundant, larger and particularly susceptible to membrane permeabilization. CADS accumulate in acidic tumours, particularly in tumour lysosomes, therefore the dose range used in vitro experiments may be relevant for the concentrations achieved after oral antihistamine use. (Study done in Denmark) Antihistamines and Ovarian Cancer Survival: Nationwide Cohort Study and in Vitro Cell Viability Assay. pmc.ncbi.nim.nih.gov

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RSO (Rick Simpson cannabis oil). Rick Simpson, who lived in Canada, brought it to everyone's attention, that cannabis may be able to cure cancer. 

He gave it freely to cancer sufferers. However, the Canadian government did not approve and made life extremely difficult for him.

Highly recommended by cancer survivors but it classed as 'anecdotal' and not recognised by most of the medical profession. There has been limited research, and there are books on how it ‘cures’ cancer.

GUINEA PIG TRIAL The first month I took a low dose, my CA125 went down from 645 to 230. // 2^nd month high dose vaginal suppositories after two weeks down to 97. Four weeks, up to 115 – which may be because I had become relaxed about taking my ‘herbal’ protocol? 

UK Medical Cannabis Clinics supply legal medicinal cannabis with a doctor’s prescription. They advertise on Facebook and include www.releaf.co.uk  and CBI Medical.com. There are about 30 Medical Clinics/ suppliers in the UK. My experience with two different clinics: They cannot legally prescribe it for ‘curative’ purposes. Only for pain relief if you have already had two other forms of pain killer prescribed by your GP.

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I disliked taking RSO and do not recommend it. It made me extremely tired, sleeping about 12 hours per day, and I became slightly depressed. However, it did reduce my tumours and was preferable to chemotherapy with no long-term side effects.

RSO is very expensive. To get the high THC recommended, you can find recipes on how to make it. i.e. The Medicinal Cannabis Oil Extraction by William Bentley. (a short easy to read book). // Marijuana Killed my Cancer by Erika M. Karohs, PH.D. (Step by step guide) // Rick Simpson Oil. Nature’s Answer for Cancer by Rick Simpson.

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CANNABIS RAW LEAVES GUINEA PIG TRIAL (uncooked and not heat treated) I am eating in salads from 26^th July 2025. I am fortunate to have a friend who grows it in his garden for his own personal medicinal use, and he provides me (free) with raw leaves.

(Info edited from ChatGPT) Eating raw cannabis leaves may offer some health benefits, though research is still limited. These benefits come mainly from the non-psychoactive compounds, particularly cannabinoid acids, terpenes, and nutrients. Raw leaves contain THCA (tetrahydrocannabinol acid) and CBDA (cannabidiol acid). They do not cause a high but may have the following benefits.

Anti-inflammatory

Neuroprotective

Anti-nausea (especially CBDA)

Potential anti-cancer properties (preliminary evidence)

Cannabis leaves are leafy greens, so they contain fibre, vitamins, (including C, K, and some B vitamins.) Minerals (iron, calcium magnesium). Chlorophyll (may support detox and blood health).

Terpenes and Flavonoids.

The leaves contain aromatic compounds (terpenes) and flavonoids which can provide antioxidant support. Help reduce oxidative stress and modulate inflammation.

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(edited from National Library of Medicine) pmc.ncbi.nim.hih.gov

COULD CANNABINOIDS PROVIDE A NEW HOPE FOR OVARIAN CANCER PATIENTS? 

(RAW CANNABIS LEAVES) Research has revealed that cannabinoids also exert anti-cancer activity such as anti-proliferative and pro-apoptotic effects through a variety of mechanisms. There is significant value in the development of these compounds as anti-cancer therapies in clinical practice as they do not produce typical toxic side effects that exist with conventional therapies. Recent clinical trials have shown their great tolerability by patients at high doses. Therefore, non-psychoactive cannabinoids are attracting pharmacological interest. Recent studies have focussed on non-psychoactive cannabinoids in ovarian cancer and have revealed promising pre-clinical results that indicate these compounds may have potential benefits. However, there are still unanswered questions and research gaps that need to be addressed.

7) CANNABINOIDS AND THEIR ANTI-TUMOUR EFFECTS.

Cannabinoids have an established role in exerting palliative effects in cancer patients and have been used to alleviate nausea, vomiting, pain and to help stimulate appetite. Besides from providing symptomatic treatment for aner patients, cannabinoids have been shown to exert anti-tumour actions through modulation of the intracellular signalling pathway implicated in cancer progression. Can exert anti-tumour effects directly through the inhibition of cell proliferation and induction of apoptosis or indirectly through the inhibition of angiogenesis, invasion, and metastasis.

Numerous studies using synthetic/endo-Phyto-cannabinoids and ECS regulators I various cancer cell lines support this notion.

Synthetic cannabinoids such as WIN-55 have also shown anti-proliferative effects on tumour progression. Currently, only Sativex and Epidiolex are the only cannabinoid-based mediations that are licensed for use in the UK. (Trials should be initiated to re-purpose Sativex and Epidiolex in ovarian cancer).

With the encouraging pre-clinical results on anti-tumour effects of cannabinoids in ovarian cancer, further research is needed. 

(Personal note: Why are big pharma trying to make artificial cannabis, rather than using one naturally grown and proven over centuries to be effective in treating a variety of ailments, including cancers?)

##(There is much more information on this site including the history and treatment of many other types of cancer with raw cannabis leaves / cannabinoids, CBD etc.) (edited from National Library of Medicine) pmc.ncbi.nim.hih.gov

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THE NUTIRITIONAL VALUE OF RAW CANNABIS. www.crxnag.com/issues/2

(edited down) Cannabis is receiving attention as a potent plant-based medicine and a powerful plant-based nutrient source. 33 states and Washington, D.C., operate medical cannabis programs and 11 states and Washington, D.C., allow for recreational adult cannabis use. Patients are rapidly gaining access to the cannabis plant. Many medical and recreational programs now allow for home cultivation.

The widespread acceptance of the cannabis plant has provided us with the opportunity to consider cannabis not only as a medicine but also as a superfood. Researchers re just beginning to explore the powerful health benefits in its raw form and understand how it may play a pivotal role in the real of plant-based nutrition.

William L. Courtney, MD, the leading expert in raw cannabis, believes it may be one of the most nutrient-dense plants to exist and raw cannabis leaves and buds provide many of the nutrients believed to be vital for a healthy life. He speculates that the raw version activates the endocannabinoid system more effectively. Many believe consuming raw cannabis is significantly less harmful and more beneficial than smoking. It presents a low chance of inducing an intoxicating experience. This allows for greater dosages without having unwanted side effects.

Raw cannabis and flowers could be considered one of the most nutrient-dense foods on the planet.  Contains essential acids, nine essential amino acid, dietary fibre, enzymes, vitamins, minerals, flavonoids, carotenoids, terpenes, and Phyto cannabinoid acids. 

Raw cannabis leave, stems, stalks and seed can provide the body with almost all the essential nutrients include carbohydrates, protein, fat, water, vitamins, minerals, trace amounts of calcium sodium potassium and omega-3 fatty acids.  

A lack of factual nutrition information is likely due to the current illegal status of cannabis but may also be attributed to the fact that there are vast compositional differences among both plants and locations the samples are taken from. Different strains of plant can contain varying quantities of phytocannaboid acids and phytonutrients. The flowering tops and leaves of the female cannabis plant contain significantly greater quantities of Phyto cannabinoid acids than do the stalks or stems. 

Fresh leaves can be used in the same manner as other ark green leafy vegetables, such as spinach, included in a salad or juiced.

There are very few precautions about safety and side effects, but important to consume only clean leaves.

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Cannabis Plants are an annual and easily grown in the UK outside in a sunny protected spot in the ground or in large pots. Indica seeds are available to order online.

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Sour Sop – Graviola. Laboratory studies show can kill some types of cancer.  I buy the pure fruit and pour it on top of blueberries. SOUR SOP – Graviola. (Expensive to buy). In laboratory studies, Graviola extracts can kill some types of cancer cells, but there have not been any studies in humans. Many sites advertise and promote Graviola as a cancer cure. Studies on animals found that Graviola may lower blood sugar and blood pressure. 2015 systematic review found that several studies show positive results. But there still needs to be more clinical trials to test.  // 2018 review found that Graviola can be used as a chemo preventative agent. (I personally, didn't feel there was any benefit. There is a lot of promotion on Facebook - so could be advertising hype? From May onwards, stopped taking it).

QUERCETIN – (Available from Nature’s Best. 500 mg or British Supplements) has shown great anti-cancer and anti-inflammatory properties. I take 2 in the morning and 2 in the evening with meals. Should not be taken if having chemotherapy.

QUERCETIN – (flavonoid, found in many fruits, veg, seeds, red onions, kale etc. Sophora Japonica has shown great anti-cancer and anti-inflammatory properties. Despite obtaining good results, both in vitro and in vivo, few clinical studies have assessed the anti-cancer effects of quercetin particularly in ovarian cancer. Quercetin possess a cytotoxic impact on ovarian cancer cells. https://pmc.ncbi.nim.nih.gov

Quercetin has been shown to inhibit the proliferation of a wide range of cancers by causing apoptosis and /or cell cycle arrest.

 VITAMIN D3  - from many suppliers. Nature’s Best: 1000 iu (25 ug) x2 taking with meals (twice a day). Not taking during the summer when I get sunshine each day.

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Turmeric Curcumin.  (20,000 mg from Nature’s Best or British Supplements). Anticancer potential by promoting cell apoptosis, suppressing cell cycle progression, inducing autophagy, inhibiting tumour metastasis. I am taking 2 in the morning and 2 in the evening with meals. 

TURMERIC CURCUMIN (20,000 mg from Nature’s Best.)

Curcumin can delay ovarian cancer, increase its sensitivity to chemotherapy, and reduce chemotherapy drugs’ side effects. Anti-cancer potential by promoting cell apoptosis, suppressing cell cycle progression, inducing autophagy, inhibiting tumour metastasis, and regulating enzyme activity. // How curcumin affects autophagy in tumour cells is not very clear. In different types of malignant diseases, curcumin regulates autophagy differently. Liu et al, found that curcumin significantly induce LC3, ATG3 and Beclin1 expresion dose-dependent manner, and increase protective autophagy in OC cells. And further clarify that the underlying mechanism of this effect is through inhibiting the AAKT/mTOR/p7056K signalling pathway. After exposure to curcumin, the autophagic flux of OC cells increased in a dosage-dependent manner. Curcumin combined with the autophagy inhibitor chloroquine (CQ) or the knockdown of LC3B by siRNA significantly enhanced the sensitivity of cells toward curcumin treatment. These results revealed that autophagy induced by curcumin protects cancer cells from death. Simultaneously, Quetal found that curcumin’s new aminocarbonyl analog B19 can induce OC cells death of apoptosis and autophagy. Apoptosis significantly increased after combined treatment with the curcumin and autophagy inhibitor 3-MA. Therefore, the combination of autophagy inhibitors and curcumin is expected to solve the resistance of curcumin to OC. 

Effects of CURCUMIN. Can inhibit tumour metastasis, includes cell invasion adhesion and angiogenesis. Shown that it inhibits metastasis in Ovarian Cancer cells by modulating several molecular targets Chemotherapy: (failure of treatment on OC). Curcumin overcomes Multi Drug Resistance through multiple mechanisms. Pre-treatment with curcumin not only decreased the expression and transcriptional activity of B-catenin, but also down regulated expression of BCl-XL etc.

CURCUMIN Recent studies found that curcumin’s anti-cancer properties are related to DNA methylation.

Although curcumin shows abundant pharmacological activity, its instability, poor water solubility, and low bioavailability still limit its application in clinical treatment.  The application of nanotechnology can increase bioavailability of curcumin by increasing the penetration of small intestine, avoiding the degradation of the intestinal condition, etc. 

Curcumin has the ability to weaken the adhesion and invasion of EOC speres to the extracellular matrix and mesothelial monolayer, thereby inhibiting metastasis. A curcumin analogue GO-Y030 depletes cancer stem cells by inhibiting the interaction between HSP70/HSP50 complex and its substrates.  https://pmc.ncbi.nim.nih.gov 

CURCUMIN is thought to have antioxidant properties which means it may decrease swelling and inflammation. It’s being explored as a cancer treatment in part because inflammation appears to play a role in cancer. Laboratory and animal research suggests that curcumin may prevent cancer, slow the spread of cancer, make chemotherapy more effective and protect healthy cells from damage by radiation therapy.  Mayo Clinic.

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ANTI-CANCER HERBS, SPICES ETC.

Green Tea, Ginger, Parsley, Cayenne Pepper, Ceylon Cinnamon, Limone, Garlic, Black Garlic, Shitake mushrooms, Cloves, Fenugreek seeds, Thymoquinone from Nigella Sativa, etc.

____________________

FROM 19^th July 2025.

Added these to my 'diet'. 

STATIN – Atorvastatin (provided by a friend – not a doctor). Would be classed as a re-purposed drug. Starting to take one a day after breakfast from 19^th July 2025.

Research: Has shown potential anti-cancer effects including against ovarian cancer, in both in vitro and in vivo studies. (See below – various other statins - ‘mouse’ trials show them to be very effective for anti-ovarian cancer.)

Atorvastatin works by inhibiting HMG-CoA reductase, reducing cholesterol synthesis and lowering levels of isoprenoids like geranylgeranyl pyrophosphate (GGPP).  

This inhibition can block protein prenylation, a process that some cancer cells rely on for survival and proliferation.

Geranylgeraniol, GGOH, when consumed, may bypass this blockage by restoring prenylation.

Concerns: There is experimental evidence (mostly in lab/animal models) suggesting that GGOH may reduce the anti-cancer effects of statins, because it can rescue cancer cells from statin-induced apoptosis.  If you are using Atorvastatin for its potential anti-cancer effects, consuming significant amounts of GGOH may reduce its benefit.

OILS containing geranylgeraniol:

Olive Oil (okay - moderate use) Risk Level Low. 

High GGOH oils to avoid include palm, sunflower and grapeseed: Moderate to High risk. 

From 20^th July – Additional herbs/supplements to add to my armoury of things cancer cells dislike, as recommended by chatGPT as anti-ovarian cancer.

REISHI (from mushroom) supplement  

GENUINE TURKEY TAIL SUPPLEMENT (from mushroom) 

GENUINE ASHWANGANDHA 

GREEN TEA EXTRACT (I can’t drink tea (or wine) due to sulphites in them).

 FURTHER RESEARCH NOTES BELOW

I AM NOT CURRENTLY USING THESE BUT NOTED FOR FUTURE REFERENCE

APRICOT KERNELS recommended by some cancer patients. They contain 2 molecules of glucose, 1 cyanide, 1 benzaldehyde. The glucose is taken by cancer cells, and they also take in the poisons! (I haven’t done any research yet, but I am saving apricot kernels as last resort).  

Artemisinin – potent antimalarial drug derived from the sweet wormwood plant. Artemisia annua. Foundation for the most effective malaria treatments. Anti-cancer research: Reduces cell proliferation and induces apoptosis in neuroblastoma. Inhibit cell growth in human ovarian cancer. Might affect blood sugar levels. 

INTERMITTENT FASTING is something some cancer patients do on a regular basis to starve their cancer cells. I did try a 24 hour fast and was okay. Others do it for 3+ days. If my cancer progresses and my tumours increase, this is something I will do more research on. However, as I have lost about 7-8 kilos, and I wasn’t ‘large’ to begin with, I am reluctant to fast and lose more weight. 

3 day fasting while taking chemotherapy is supposed to be beneficial. Fasting the day before, during, and the day after while your cancer cells are compromised. 

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IMMUNO CANCER THERAPY

Dendritic Cell Therapy. Available at some clinics. May cost approx. £30,000 for a package that includes diet etc. // Take blood from you, and then intravenously give back your blood with cells that recognise cancer cells and destroy them. // This treatment is only about 5% - !5% effective for ovarian cancer, and probably only is successful for about two years? More research needed. 

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CIMETIDINE Brand name Tagamet. Histamine H2 receptor, treats heartburn and peptic ulcers. Small trial, 28 advanced serous ovarian cancer patients, chemo augmented with CIM at a dose of 800mg/day, 2 weeks before surgery and continuing with chemo showed significant improvements in overall survival. Inhibits cancer cell adhesion to endothelial cells and prevents metastasis by blocking E-selectin expression. 

CHLOROQUINE (used to treat malaria). Expected to have diverse effects on cancer progression. Inhibition of late autophagy can affect cell survival, as well as antigen processing and presentation in the immune system. The chloroquine family of drugs have been used for the prevention and treatment of malaria. Autophagy is an intrinsic cellular mechanism by which cells degrade and recycle their dysfunctional components through lysosomes. Contrasting roles of autophagy has been reported in cancer cells. It may function as a tumour suppressor mechanism in cells by degrading damaged proteins and organelles and therefore preventing their accumulation in the cells. However, it can also promote cell survival by recycling intracellular organelles and proteins in tumours. Hence autophagy is considered a mechanism potential leading to invasion, chemotaxis, trans-differentiation and clonogenicity of tumour cells.

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HIGH DOSE VITAMIN C intravenously IVC, (not orally).

It turns from an antioxidant into a pro-oxidant giving off free oxygen to the area around a tumour. The reaction oxygen species (ROS) is hydrogen peroxide (H202). This oxygen production near the cell causes a kind of ‘rusting’ and allows the cell to be attacked and killed. ‘Mounting evidence indicates that Vit C has potential to be a potent anti-cancer agent when administered intravenously in high doses. Early clinical trials have confirmed safety and indicated efficacy in eradicating tumour cells of various cancer types.   Moreover, high dose IVC is powerful as an adjuvant treatment for cancer, acting synergistically with many standard (chemo) therapies, as well as a method for mitigating the toxic side-effects of chemotherapy.– Journal of Experimental & Clinical Cancer Research.

(There are pages of information on Intravenous Vit. C. IVC – both positive and negative). 

Based on the observations that the combination of parenteral ascorbate with carboplatin and paclitaxel, synergistically inhibited ovarian cancer in preclinical ovarian cancer models. Ma et al conducted clinical trial assessing effectiveness and toxicity of such treatment with stage III and IV ovarian cancer.  The median time for disease progression was 8.75 months longer with IVC. // Ascorbic acid added to paclitaxel/carboplatin therapy in stage II and IV ovarian cancer patients reduced chemotherapy induced toxicity. IVC did not increase the rate of graded 3 or 4 toxicities. Also not observed grade 5 treatment -related toxicities (death). Also, authors proved that grade 1 and grade 2 toxicities i.e. neurotoxicity, bone marrow, infections, hepatobiliary/pancreatic toxicity, dermatological toxicity, and toxicities in the skin, renal genitourinary, pulmonary and gastrointestinal systems, were significantly decreased in the group received ascorbic acid, when compared to sole chemotherapy. Possible side effects during IVC administration, alleviated same day as infusion:  Mild light- headedness and nausea. Thirst, dry mouth and skin, increased urinary flow, diarrhoea, headache. pmc.ncbi.nim.nih.gov.

Personal Note - I have made enquiries about receiving Intravenous Vit. C from a clinic, but I will need a prescription either from my GP or my oncologist. As neither believe in alternative therapies, and that the only treatment I should receive is chemotherapy, this may be difficult to obtain, should I feel I need it.

 MILK THISTLE (Silybum marianum), particularly its active compound silymarin, has shown anti-ovarian cancer potential in several preclinical studies. Inhibition of ovarian caner cell growth. (e.g. SKOV3, OVCAR3 cell lines). Induction of apoptosis (programmed cancer cell death). Suppression of metastasis and angiogenesis (formation of new blood vessels that feed tumours).  No human trials. Has been used as a supportive therapy to protect the liver during chemotherapy. (Research edited from ChatGPT.)

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MORINGA oleifera has shown to have potential anti-cancer properties, including effects against ovarian cancer, although more research is needed. Several studies demonstrated that extracts from Moringa (especially from leaves, seeds, and bark) can inhibit the growth of ovarian cancer cells. Induce apoptosis (programmed cell death) of caner cells. Reduce oxidative stress and inflammation both of which play roles in cancer progression. (Research edited from CHAPgpt – much more available. No human trials).

MISTLETOE Therapy - I was offered this treatment by a London clinic to help with my cancer. (very expensive). Not permitted in the USA. I couldn't see evidence/research to support me receiving it.

1)Randomized and non-randomized studies. 

Mistletoe extracts in addition to basic oncological treatments, had a positive effect on survival time with patients with ovarian cancer and solid cancers. The efficacy is still controversial. 

Injectable, not available commercially in the USA and not approved as a treatment for people with cancer. Is available as capsule/powder form in the UK. Or Wellness Clinic – injections.

Trial: Randomised studies: Mistletoe extracts Iscador might prolong overall survival of ovarian cancer patients.   Pubmed.ncbi.nim.nih.gov

2) Systematic review: Mistletoe - Quality of Life and toxicity Sept & Oct 2017.

Search identified 3647 articles and 28 publications. 2639 patients included in review. 

In nearly all studies, mistletoe was added to conventional therapy. 

Regarding quality of life, 17 publications reported results. Studies with better methodological quality show less or no effects on quality of life.

Conclusions: Quality of Life or reduction of treatment – associated side effects, a thorough review of the literature does not provide any indication to prescribe mistletoe to patients with cancer.

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METHYLENE BLUE METABOLIC THERAPY

Restrains in Vivo Ovarian Tumour Growth (in petri dish)

Study confirms the potential of methylene blue (MB) in a treatment strategy for ovarian cancer. Findings revealed MBs ability to impede ovarian cancer progression, especially against tumours showing resistance to conventional chemotherapies (carboplatin).

ChatGPT research – edited below.

1.    Sonodynamic Therapy (SDT) in vitro. 2010 study applied 100pM MB plus ultrasound (0.46 W/com2) to ovarian HO-8910 cells. This combination triggered significantly increased apoptosis, (early & late), heightened RS, and nuclear condensation – far more than MB or ultrasound alone. – There is a lot more research and information. It would be wise to monitor known toxicity at high doses and keep an eye out for emerging early-phase trials. (No direct ovarian cancer trials yet). 

NICOLSAMIDE oral anthelminthic. (overcomes cisplatin resistance in human ovarian cancer). To treat tapeworms, anti-cancer. Effectively inhibits cell proliferation, supresses cell migration and induces cell apoptosis in cisplatin-resistant ovarian cancer cells at a very low micromole range. Further demonstrated NA effectively inhibited xenograft tumour growth of the SKOV3CR cells.

 

WEIGHT LOSS INJECTIONS could help prevent cancer and have anti-cancer effect. (My Question – is this because they reduce absorption of sugars and fats and starve cancer cells????)

STATINS – re-purposed drugs

Simvastatin. Several epidemiological studies and in vitro testing have demonstrated that statins have an anti-cancer effect, especially on ovarian cancer. … The mutation rate of TP53 in ovarian cancer is as high as 95% while HMGCR is often highly expressed in TP53 mutant tumours.  It seems unrealistic for a lipid-lowering drug to completely inhibit tumour growth. Therefore, statins play more synergistic roles in the treatment of ovarian cancer. pmc.ncbi.nim.nih.gov  

STATIN - Pitavastatin can inhibit the growth of ovarian cancer xenografts but only if the diet Is modified to eliminate geranylgeraniol (found in edible oils such as linseed and sunflower. . 

STATIN - BESTATIN – more research has found this is effective against ovarian cancer cells 

STATIN - PITAVASTATIN + IVERMECTIN

Ivermectin augments the anti-cancer activity of Pitavastatin in Ovarian Cancer Cells 

By Mohammed Jasmin Jaward and Alan Richardson. The School of Pharmacy and Bioengineering, Keele Uni. // College of Veterinary Medicine, Kerbala Uni.

“We have previously shown that pitavastatin has the potential to be used to treat ovarian cancer, although relatively high doses are likely to be necessary.  One solution is to identify drugs that are synergistic with pitavastatin. Here, tested with anti-parasitic drug ivermectin in six ovarian cancer cell lines. When tested on its own, ivermectin inhibited the growth of the cells but only with modest potency. When combined, ivermectin showed synergy with pitavastatin in 3 cell lines, most evident in COV-318 cells.

We and many others have explored the potential for statins to be repurposed for use as anti-cancer agents. Numerous laboratory studies have shown statins exert cytotoxic effects against cancer cells.

We have shown that one statin, pitavastatin can inhibit the growth of ovarian cancer xenografts, but only if the diet is modified to eliminate geranylgeraniol which otherwise bypasses the block ade of hydroxymethyl glutaryl coenzyme.

Despite robust preclinical data showing that statins have anti-cancer activity, statins have yet to show significant activity in clinical trials. “We have argued that this reflect the poor design of clinical trials which have uniformly failed to address all the factors we predict influence the clinical activity of statins. This failure includes poor choice of statin, inappropriate dose, inappropriate dosing interval, and failure to consider dietary sources of geranylgeraniol. Lipophilic statins are typically more potent as anti-cancer agents than hydrophilic ones.

We consider pitavastatin most likely statin to be effective as an anti-cancer agent. However, relatively high doses are likely to be required. The major adverse event is myopathy, although infrequent.

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 PRP anti-angiogenic. Combination of Trypsinogen and Chymotrypsinogen.  Bovine pancreatic pro-enzymes. Significantly inhibited advanced pancreatic and ovarian cancers. Patients lived significantly longer than expected. Treated daily with rectal suppositories. https://pmc.ncbi.nim.nih.gov.articles/pMC5656647/1 (Maybe available to purchase as oral anti-inflammatory medication. Trypsin & Chymotrypsinogen. May not be absorbed so well.)  

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 USNIC ACID

(Research NIH National Library of Medicine). (There is quite a lot of research, and these notes have been edited from version on Facebook Page - Healing Cancer Study.  

USNIC ACID (UA) from Usnea lichens demonstrates a rare breadth of anti-cancer actions. It disrupts cancer cell growth, triggers apoptosis and autophagy, block angiogenesis and metastasis, and modulates immune and inflammatory pathways.

UA’s selectivity for cancer cells over normal cells, and its ability to target multiple cancer hallmarks simultaneously, make it a uniquely promising adjunct to integrative oncology. 

METABOLIC DISRUPTION.  UA uncouples mitochondrial function and impairs glycolysis placing metabolic stress on cancer cells.

PROTEIN-PROTEIN INTERACTIONS: Recent research reveals that UA binds to and degrades 14-3-3 proteins, master regulators of cancer cell signalling, metabolism, and survival. This is a novel and potentially transformative mechanism, as 14-3-3 proteins are central to cancer and fungal pathogen biology.

SYNERGY WITH STANDARD THERAPIES: UA enhances the effects of chemotherapeutics like paclitaxel and cetuximab, primarily through autophagy modulation and mitochondrial stress. Its ability to overcome drug resistance pathways and sensitise cancer cells to apoptosis is a key advantage. 

SAFETY, DOSING AND PRACTICAL USE.

UA has outperformed standard chemotherapies (e.g. 5-fluorouracil, cyclophosphamide) with less toxicity in animal models.

Hepatoxicity Context: Serious liver injury is linked to high-dose diet pills, not traditional or research use. UA is much safer at lower controlled doses, but monitoring is essential. 

Dosing: Typical tincture dosing is 1-2 mL (30-60 drops) 2 – 4 times daily, up to 3 weeks cycle. Most tinctures are not standardised for UA content; research extracts may contain 0.24-0.6 mg/mL. 

INTERACTIONS: Strong antioxidants (NAC, high dose Vit C, E etc) may blunt UA’s effects. Fasting-mimicking diets and agents like sodium selenite for IGF-1 inhibitions and Lipsomal Shikonin may enhance UA’s impact by targeting cancer’s escape survival and growth pathways. 

UA should be used as an adjunct to standard oncology care, not as a replacement.  Protocols should be tailored to individual needs. 

BOTTOM LINE

Usnic Acid is a powerful, multi-targeted natural agent with broad anti-cancer and anti-fungal properties, unique mechanisms, and a favourable safety profile at appropriate doses.

Its ability to disrupt cancer metabolism, target protein-protein interactions, and synergise with standard therapies makes it a valuable tool in integrative oncology. 

FACEBOOK NOTE BY MARK LINTERN - re USNIC ACID.

14-3-3 proteins are specific to Eukaryotic cells and not prokaryotic cells, so this indicates that the potential dual effect it has against cancer could be primarily due to its ability to affect both the ell itself, and the fungal pathogen I propose is driving the disease. Although Usnic acid is also anti-bacterial – by disrupting bacterial RNA and DNA synthesis. 

Having said that, it only appears to affect Gram-positive bacteria. So, if cancer was caused by bacteria Usnic acid’s anti-cancer action suggests that only GRAM- positive bacteria could cause the disease. The issue I have with this is that prolonged use of anti-biotics presents with a dose-dependent increased risk for cancer progression – suggesting that bacteria are not the micro-organism directly responsible for driving the disease. This of course brings us back to fungi in this thread. I’m not aware of how Usnic acid affects parasites (helminths) or viruses though.

 

BOOKS

THE CANCER RESOLUTION, by Mark Lintern (available on Amazon). A new cancer theory. You can listen to interviews with him on You Tube. Well worth a ‘listen’. I have purchased his book – which is ‘big’ read.

 

HOW TO STARVE CANCER by Jane McClelland. (Highly recommended. THE MOST helpful read.) 

(Not recommended: Curing the Incurable? Fenbendazole, Ivermectin, and the Cancer Question by Sarah Alton, MSC. Contains a lot of waffle and very little about either Fenbendazole or Ivermectin, or cancer.)

 EXPLANATION OF LONG – MEDICAL WORDS

 APOPTOSIS A type of cell death in which a series of molecular steps in a cell lead to its death. The process may be blocked in cancer cells.

AUTOPHAGY is the natural conserved degradation of the cell that removes unnecessary or dysfunctional components through lysosome-dependent regulated mechanism.

CYTOTOXIC EFFECTS how harmful a substance is to an organism – to cells. Can cause cell damage or death, either through necrosis or apoptosis. 

Glycolysis – METABOLIC PATHWAY THAT CONVERS GLUCOSE INTO PYRUVATE AND, IN MOST ORGANISMS, OCCURS IN THE LIQUID PART OF CELLS. The free energy released I this process is used to form the high energy molecules adenosine triphosphate and reduced nicotinamide adenine dinucleotide.

 Metastatic Cancer – Cancer that has spread from where it started. For many types of cancer, it is also called stage 4 cancer.  

Necrosis is the death of body tissue. It occurs when too little blood flows to the tissue. This can be from injury, radiation or chemicals. Necrosis cannot be reversed. Can occur due to injuries, infections or diseases. 

Xenograft: Patient derived xenografts are models of cancer where the tissue or cells from a patient’s tumour are implanted into an immunodeficient or humanized mouse.

 (Good luck and please do share your research)

 

Email: susan.k.moore@btinternet.com

OTHER PEOPLE’S RESEARCH and successes.

Noel Watson- Facebook Healing Cancer Study Support Group  

Diagnosed with pancreatic cancer 14^th Aug 2020. Gave up chemotherapy 27 months ago. Is stable, tumour size 2.5cm no metastasis.

Asked ChatGPT for the 10 best products in ‘his’ protocol. 

1.    Ivermectin 48mg daily.  // Why: Potent anticancer, anti-inflammatory, anti-parasitic, disrupts WNT/b-catenin and PAK1 pathways involved in pancreatic cancer. High repurposing potential. Bonus – crosses the blook-brain barrier and supports microglial detox.

 I hope the information I have provided is helpful. 

Welcome to my journey 

My research notes are free of copyright, and you may share

Website: www.susan-moore.co.uk  

Facebook page: Susan Moore also Susan’s Cancer Journey

Email: susan.k.moore@btinternet.com

BOOK - ENCHANTED CASTLE - by S.K. Moore, profits to children's charities. 

You may wonder, why not donate to cancer charities? 

Personally, I cannot see what actual independent 'human' trials the cancer research charities are supporting with regard to natural therapies with less toxic outcomes. It is generally agreed, cancer treatment outcomes have not improved much over the past 50 years. Too many people are suffering.

We need more 'human' trials on alternative treatments, repurposed drugs etc. Without it, doctors are unable to support/help independents, like myself, who do not wish to take chemotherapy. 

We also need doctors, oncologists, alternative therapists, to gain knowledge and expertise so they can advise and support patients. My GP told me flatly she only believed in medicine that had been scientifically proven, and not in any complimentary medicines. 

While big pharma are making millions/billion out of selling toxic drugs, they are not investing in less disagreeable 'cures' or treatments.